By Guilleminault C. (ed.)

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2002) Timecourses in renin and blood pressure during sleep in humans. J. , 11(1): 73–79. Chihara, K, Kato, Y, Maeda, K, et al. (1976) Suppression by cyproheptadine of human growth hormone and cortisol secretion during sleep. J. Clin. , 57(6): 1393– 1402. Chokroverty, S (1980) Phasic tongue movements in human rapid eye-movement sleep. Neurology, 30(6): 665–668. Cianci, T, Zoccoli, G, Lenzi, P and Franzini, C (1990) Regional splanchnic blood flow during sleep in the rabbit. , 415(5): 594–597. Conway, J, Boon, N, Jones, JV and Sleight, P (1983) Involvement of the baroreceptor reflexes in the changes in blood pressure with sleep and mental arousal.

1A. All channel calibration is shown. All amplifiers have the same sensitivity and high- and low-frequency filter settings. ity, high-frequency filter and low-frequency filter settings and a known signal is sent through all amplifiers simultaneously. 1A). A second calibration is performed for the specific study protocol. 1B). The protocol calibration ensures that all amplifiers are set to ideal conditions for recording the parameter of interest. Filter and sensitivity settings should be clearly documented for each channel.

The EEG channels in the trace are C3/A2 and O2/A1. Since the artifact does not appear in the O2/A1 channel the artifact is localized to the LOC electrode. The electrode placement may be insecure or the patient may be lying on the electrode and producing movement of the LOC electrode in association with breathing. Additional artifact is noted in the EMG channel. This signal is contaminated with ECG artifact and the intermittent slower activity as well as the wandering baseline are most likely due to a loose lead.

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