By Maria C. Linder

Copper has lengthy been often called necessary to dwelling structures, partially via its basic position in electron shipping and respiratory. through the years into the current, its involvement in an ever expanding variety of approaches in every kind of organisms has turn into obvious, and new and fascinating vistas of its roles in such components because the significant fearful method, and in humoral features, are showing at the horizon. even if the biochemistry of this point has no longer been studied approximately up to that of many others, a for­ midable quantity of labor has been conducted. It has hence been a problem to provide a precis of what has been stumbled on that gives either breadth and intensity. My target has been to attempt to be as entire as attainable, inside a few obstacles. i've got attempted to supply uncomplicated info and simple information that are supposed to stay helpful for a very long time. The objective has additionally been to interpret the place we at the moment stand in our wisdom of the constitution, functionality, legislation, and metabolism of Cu-dependent procedures and sub­ stances, specially proteins. therefore, i've got attempted to make this a resource ebook for old in addition to present info on all elements of copper bio­ chemistry, and a precis of our present wisdom of copper-dependent proteins and tactics. lots of the study on copper has been performed on vertebrates, specially mammals. This has performed a job within the association of the book.

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Extra resources for Biochemistry of Copper

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Clearly, more work is needed to understand exactly what is the effect of fructose on copper metabolism. The most obvious alternative, namely, that fructose enhances Cu excretion, should be checked (after injection of radioisotope). , 1989). In the copper studies of Fields (1986d, 1987), this sugar alcohol was produced in large (and probably damaging) quantities by male (but not female) rats on 60% fructose diets, and copper deficiency was also enhanced by fructose only in the males. This implies that sorbitol itself may be damaging tissue parts vital to copper absorption, distribution, or excretion.

Reprinted, with permission, from Suzuki (1980). (B) Electrophoretic migration of radioactive copper-binding components obtained in the metallothionein peak on Sephadex G-75, derived from duodenal mucosal cytosol of rats given 150 Jl8 of Cu ( 64Cu) or trace ( 67Cu) doses of copper by intragastric intubation (T. Sonsma and M. C. Linder, unpublished results). 65 Rf peaks were also labeled by 65Zn(II), 109Cd(II), and P5S] Cys. 40 Chapter 2 metallothionein-like component (Fig. 2-5C), while at higher doses given in vitro (see also Table 2-8), binding to the high-molecular-weight component(s) of 80,000 Da or more becomes important (Fig.

The most obvious alternative, namely, that fructose enhances Cu excretion, should be checked (after injection of radioisotope). , 1989). In the copper studies of Fields (1986d, 1987), this sugar alcohol was produced in large (and probably damaging) quantities by male (but not female) rats on 60% fructose diets, and copper deficiency was also enhanced by fructose only in the males. This implies that sorbitol itself may be damaging tissue parts vital to copper absorption, distribution, or excretion.

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